All Is Well: Heal Your Body With Medicine, Affirmations, and Intuition (40 page)

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Clinical and Experimental Immunology
84, no. 3 (June 1991):

440–444.

3. T.G. Digan and J.F. Cryan, “Regulation of the Stress Response by

the Gut Microbiota: Implications for Psychoneuroendocrinology
,”

Psychoneuroendocrinology
37, no. 9 (September 2012): 1369–1378;

G.B. Glavin, “Restraint Ulcer: History, Current Research and Future

Implications,”
Brain Research Bulletin
Supplement, no. 5 (1980): 51–58.

4. J.M. Lackner et al., “Self Administered Cognitive Behavior Therapy for

Moderate to Severe IBS: Clinical Efficacy, Tolerability, Feasibility,”
Clinical

Gastroenterology and Hepatology
6, no. 8 (August 2008): 899–906; F. Alexander,

“Treatment of a Case of Peptic Ulcer and Personality Disorder,”
Psychosomatic

Medicine
9, no. 5 (September 1947): 320–330; F. Alexander, “The Influence of

Psychologic Factors upon Gastro-Intestinal Disturbances: A Symposium—I.

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General Principles, Objectives, and Preliminary Results,”
Psychoanalytic

Quarterly
3 (1934): 501–539.

5. S.J. Melhorn et al., “Meal Patterns and Hypothalamic NPY Expression During

Chronic Social Stress and Recovery,”
American Journal of Physiology Regulatory,

Integrative and Comparative Physiology
299, no. 3 (July 2010): R813–R822; I.K.

Barker et al., “Observations on Spontaneous Stress-Related Mortality Among

Males of the Dasyurid Marsupial Antechinus Stuartii Macleay,”
Australian

Journal of Zoology
26, no. 3 (1978): 435–447; J.L. Barnett, “A Stress Response

in Som Antechinus Stuartii (Macleay),”
Australian Journal of Zoology
21, no.

4 (1973): 501–513; R. Ader, “Effects of Early Experience and Differential

Housing on Susceptibility to Gastric Erosions in Lesion-Susceptible Rats,”

Psychosomatic Medicine Journal of Behavioral Medicine
32, no. 6 (November

1970): 569–580.

6. G.L. Flett et al., “Perfectionism, Psychosocial Impact and Coping with

Irritable Bowel Disease: A study of Patients with Crohn’s Disease and

Ulcerative colitis,”
Journal of Health Psychology
16, no. 4 (May 2011):

561–571; P. Castelnuovo-Tedesco, “Emotional Antecedents of Perforation

of Ulcers of the Stomach and Duodenum,”
Psychosomatic Medicine
24, no. 4

(July 1962): 398–416.

7. R.K. Gundry et al., “Patterns of Gastric Acid Secretion in Patients with

Duodenal Ulcer: Correlations with Clinical and Personality Features,”

Gastroenterology
52, no. 2 (February 1967): 176–184; A. Stenback, “Gastric

Neurosis, Pre-ulcers Conflict, and Personality in Duodenal Ulcer,”
Journal of

Psychosomatic Research
4 (July 1960): 282–296; W.B. Cannon, “The Influence

of Emotional States on the Functions of the Alimentary Canal,”
The American

Journal of the Medical Sciences
137, no. 4 (April 1909): 480–486.

8. E. Fuller-Thomson et al., “Is Childhood Physical Abuse Associated with

Peptic Ulcer Disease? Findings From a Population-based Study,”
Journal of

Interpersonal Violence
26, no. 16 (November 2011): 3225–3247; E.J. Pinter

et al., “The Influence of Emotional Stress on Fat Mobilization: The Role

of Endogenous Catecholamines and the Beta Adrenergic Receptors,”
The

American Journal of the Medical Sciences
254, no. 5 (November 1967): 634–651.

9. S. Minuchin et al., “Psychosomatic Families: Anorexia Nervosa in Context,”

(Harvard University Press, 1978): 23–29; G.L. Engel, “Studies of Ulcerative

Colitis: V. Psychological Aspects and Their Implications for Treatment,”
The

American Journal of Digestive Diseases and Nutrition
3, no. 4 (April 1958): 315–

337; J.J. Groen and J.M. Van der Valk, “Psychosomatic Aspects of Ulcerative

Colitis,”
Gastroenterologia
86, no. 5 (1956): 591–608; G.L. Engel, “Studies of

Ulcerative Colitis. III. The Nature of the Psychologic Process,”
The American

Journal of Medicine
19, no. 2 (August 1955): 231–256.

10. S.J. Melhorn et al., “Meal Patterns and Hypothalamic NPY Expression

During Chronic Social Stress and Recovery,”
American Journal of Physiology-

Regulatory, Integrative and Comparative Physiology
299, no. 3 (September

2010): R813–R822; P.V. Cardon, Jr., and P.S. Mueller, “A Possible Mechanism:

Psychogenic Fat Mobilization,”
Annals of the New York Academy of Sciences

125 (January 1966): 924–927; P.V. Cardon, Jr., and R.S. Gordon, “Rapid

228

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Endnotes

Increase of Plasma Unesterified Fatty Acids in Man during Fear,”
Journal of

Psychosomatic Research
4 (August 1959): 5–9; M.D. Bogdonoff et al., “Acute

Effect of Psychologic Stimuli upon Plasma Non-esterified Fatty Acid Level,”

Experimental Biology and Medicine
100, no. 3 (March 1959): 503–504.

11. R.N. Melmed et al., “The Influence of Emotional State on the Mobilization

of Marginal Pool Leukocytes after Insulin-Induced Hypoglycemia. A Possible

Role for Eicosanoids as Major Mediators of Psychosomatic Processes,”
Annals

of the New York Academy of Sciences
496 (May 1987): 467–476; H. Rosen and

T. Lidz, “Emotional Factors in the Precipitation of Recurrent Diabetic

Acidosis,”
Psychosomatic Medicine Journal of Behavioral Medicine
11, no. 4

(July 1949): 211–215; A. Meyer et al., “Correlation between Emotions and

Carbohydrate Metabolism in Two Cases of Diabetes Mellitus,”
Psychosomatic

Medicine Journal of Behavioral Medicine
7, no. 6 (November 1945): 335–341.

12. S.O. Fetissov and P. Déchelotte, “The New Link between Gut-Brain Axis

and Neuropsychiatric Disorders,”
Current Opinion in Clinical Nutrition and

Metabolic Care
14, no. 5 (September 2011): 477–482; D. Giugliano et al., “The

Effects of Diet on Inflammation: Emphasis on the Metabolic Syndrome,”

Journal of the American College of Cardiology
48, no. 4 (August 2006): 677–

685; G. Seematter et al., “Stress and Metabolism,”
Metabolic Syndrome and

Related Disorders
3, no. 1 (2005): 8–3; A.M. Jacobson and J.B. Leibovitch,

“Psychological Issues in Diabetes Mellitus,”
Psychosomatics: Journal of

Consultation Liaison Psychiatry
25, no. 1 (January 1984): 7–15; S.L. Werkman

and E.S. Greenberg, “Personality and Interest Patterns in Obese Adolescent

Girls,”
Psychosomatic Medicine Journal of Biobehaviorial Medicine
29, no. 1

(January 1967): 72–80.

13. J.H. Fallon et al., “Hostility Differentiates the Brain Metabolic Effects of

Nicotine,”
Cognitive Brain Research
18, no. 2 (January 2004): 142–148;

R.N. Melmed et al., “The Influence of Emotional Stress on the Mobilization

of Marginal Pool Leukocytes after Insulin-Induced Hypoglycemia. A Possible

Role for Eicosanoids as Major Mediators of Psychosomatic Processes,”
Annals

of the New York Academy of Sciences
496 (May 1987): 467–476; P.V. Cardon

Jr. and P.S. Mueller, “A Possible Mechanism: Psychogenic Fat Mobilization,”

Annals of the New York Academy of Sciences
125 (January 1966): 924–927;

M.D. Bogdonoff et al., “Acute Effect of Psychologic Stimuli upon Plasma

Non-Esterified Fatty Acid Level,”
Experimental Biology and Medicine
100, no. 3

(March 1959): 503–504; P.V. Cardon, Jr., and R.S. Gordon, “Rapid Increase of

Plasma Unesterified Fatty Acids in Man during Fear,”
Journal of Psychosomatic

Research
4 (August 1959): 5–9; A. Meyer et al., “Correlation between

Emotions and Carbohydrate Metabolism in Two Cases of Diabetes Mellitus,”

Psychosomatic Medicine Journal of Behavioral Medicine
7, no. 6 (November

1945): 335–341.

Fourth Emotional Center

1. H.P. Kapfhammer, “The Relationship between Depression, Anxiety and

Heart Disease—a Psychosomatic Challenge,”
Psychiatr Danubina
23, no. 4

(December 2011): 412–424; B.H. Brummett et al., “Characteristics of Socially

229

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Isolated Patients With Coronary Artery Disease Who Are at Elevated Risk for

Mortality,”
Psychosomatic Medicine Journal of Biobehavioral Medicine
63, no. 2

(March 2001): 267–272; W.B. Cannon,
Bodily Changes in Pain, Hunger, Fear

and Rage
(New York: D. Appleton & Co., 1929).

2. K.S. Whittaker et al., “Combining Psychosocial Data to Improve Prediction

of Cardiovascular Disease Risk Factors and Events: The National Heart, Lung,

and Blood Institute–Sponsored Women’s Ischemia Syndrome Evaluation

Study,”
Psychosomatic Medicine Journal of Biobehavioral Medicine
74, no. 3

(April 2012): 263–270; A. Prasad et al., “Apical Ballooning Syndrome (Tako-

Tsubo or Stress Cardiomyopathy): A mimic of Acute Myocardial Infarction,”

American Heart Journal
155, no. 3 (March 2008): 408–417; Wittstein, I.S.

et al. “Neurohumoral Features of Myocardial Stunning Due to Sudden

Emotional Stress,”
The New England Journal of Medicine
352, no. 6 (February

2005): 539–548; M.A. Mittleman et al., “Triggering of Acute Myocardial

Infarction Onset of Episodes of Anger,”
Circulation
92 (1995): 1720–1725;

G. Ironson et al., “Effects of Anger on Left Ventricular Ejection Fraction in

Coronary Artery Disease,”
American Journal of Cardiology
70, no. 3 (August

1992): 281–285; R.D. Lane and G.E. Schwartz, “Induction of Lateralized

Sympathetic Input to the Heart by the CNS During Emotional Arousal: A

Possible Neurophysiologic Trigger of Sudden Cardiac Death,”
Psychosomatic

Medicine
49, no. 3 (May–June 1987): 274–284; S.G. Haynes et al., “The

Relationship of Psychosocial Factors to Coronary Heart Disease in the

Framingham Study. III. Eight-Year Incidence of Coronary Heart Disease,”

American Journal of Epidemiology
111, no. 1 (January 1980): 37–58.

3. T.W. Smith et al., “Hostility, Anger, Aggressiveness, and Coronary Heart

Disease: An Interpersonal Perspective on Personality, Emotion, and Health.”

Journal of Personality
72, no. 6 (December 2004): 1217–1270;

T.M. Dembroski et al., “Components of Hostility as Predictors of Sudden

Death and Myocardial Infarction in the Multiple Risk Factor Intervention

Trial,”
Psychosomatic Medicine
51, no. 5 (September–October 1989): 514–522;

K.A. Matthews et al., “Competitive Drive, Pattern A, and Coronary Heart

Disease,”
Journal of Chronic Diseases
30, no. 8 (August 1977): 489–498;

I. Pilowsky et al., “Hypertension and Personality,”
Psychosomatic Medicine
35,

no. 1 (January–February 1973): 50–56.

4. M.D. Boltwood et al., “Anger Reports Predict Coronary Artery Vasomotor

Response to Mental Stress in Atherosclerotic Segments,”
American Journal

of Cardiology
72, no. 18 (December 15, 1993): 1361–1365; P.P. Vitaliano et

al., “Plasma Lipids and Their Relationships with Psychosocial Factors in

Older Adults,”
Journal of Gerontology, Series B, Psychological Sciences and Social

Sciences
50, no. 1 (January 1995): 18–24.

5. H.S. Versey and G.A. Kaplan, “Mediation and Moderation of the Association

Between Cynical Hostility and Systolic Blood Pressure in Low-Income

Women,”
Health Education & Behavior
39, no. 2 (April 2012): 219–228.

6. P.J. Mills and J.E. Dimsdale, “Anger Suppression: Its Relationship to Beta-

Adrenergic Receptor Sensitivity and Stress-Induced Changes in Blood

Pressure,”
Psychological Medicine
23, no. 3 (August 1993): 673–678.

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Endnotes

7. M.Y. Gulec et al., “Cloninger’s Temperament and Character Dimension

of Personality in Patients with Asthma,”
International Journal of Psychiatry

in Medicine
40, no. 3 (2010): 273–287; P.M. Eng et al., “Anger Expression

and Risk of Stroke and Coronary Heart Disease Among Male Health

Professionals,”
Psychosomatic Medicine
65, no. 1 (January–February 2003):

100–110; L. Musante et al., “Potential for Hostility and Dimensions of

Anger,”
Health Psychology
8, no. 3 (l989): 343–354; M.A. Mittleman et al.,

“Triggering of Acute Myocardial Infarction Onset of Episodes of Anger,”

Circulation
92 (1995): 1720–1725; M. Koskenvuo et al., “Hostility as a Risk

Factor for Mortality and Ischemic Heart Disease in Men,”
Psychosomatic

Medicine
50, no. 4 (July–August 1988): 330–340; J.E. Williams et al,

“The Association Between Trait Anger and Incident Stroke Risk: The

Atherosclerosis Risk in Communities (ARIC) Study,”
Stroke
33, no. 1

(January 2002): 13–19; N. Lundberg et al., “Type A Behavior in Healthy

Males and Females as Related to Physiological Reactivity and Blood Lipids,”